2012, ISBN: 1461433444
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2012, ISBN: 1461433444
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2012, ISBN: 1461433444
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2012, ISBN: 9781461433446
Springer, 2012-05-23. 2012. Hardcover. Good., Springer, 2012-05-23, 2.5
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2012, ISBN: 1461433444
Gebundene Ausgabe Gesundheitswesen, Immunsystem - Immunologie - Immun - Autoimmunität, Infektiologie, Biologie / Mikrobiologie, Mikrobiologie - Mikroorganismus, Virologie, Virus - Virolo… Altro …
Minarovits, Janos (Herausgeber); Niller, Hans Helmut (Herausgeber):
Patho-Epigenetics of Disease - copertina rigida, flessible2012, ISBN: 1461433444
Gebundene Ausgabe Gesundheitswesen, Immunsystem - Immunologie - Immun - Autoimmunität, Infektiologie, Biologie / Mikrobiologie, Mikrobiologie - Mikroorganismus, Virologie, Virus - Virolo… Altro …
2012
ISBN: 1461433444
[EAN: 9781461433446], Neubuch, [PU: Springer New York 2012-05-23, New York], Language: ENG, Books
2012, ISBN: 9781461433446
Springer, 2012-05-23. 2012. Hardcover. Good., Springer, 2012-05-23, 2.5
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Informazioni dettagliate del libro - Patho-Epigenetics of Disease
EAN (ISBN-13): 9781461433446
ISBN (ISBN-10): 1461433444
Copertina rigida
Anno di pubblicazione: 2012
Editore: Springer-Verlag GmbH
465 Pagine
Peso: 0,852 kg
Lingua: Englisch
Libro nella banca dati dal 2007-07-24T02:05:42+02:00 (Rome)
Pagina di dettaglio ultima modifica in 2022-04-27T22:44:26+02:00 (Rome)
ISBN/EAN: 1461433444
ISBN - Stili di scrittura alternativi:
1-4614-3344-4, 978-1-4614-3344-6
Stili di scrittura alternativi e concetti di ricerca simili:
Autore del libro : minar, helmut, nill, niller
Titolo del libro: ostberlin, epigenetics and disease
Dati dell'editore
Autore: Janos Minarovits; Hans Helmut Niller
Titolo: Patho-Epigenetics of Disease
Editore: Springer; Springer US
466 Pagine
Anno di pubblicazione: 2012-05-23
New York; NY; US
Stampato / Fatto in
Peso: 0,881 kg
Lingua: Inglese
160,49 € (DE)
164,99 € (AT)
177,00 CHF (CH)
POD
XIV, 466 p.
BB; Medical Microbiology; Hardcover, Softcover / Medizin/Nichtklinische Fächer; Mikrobiologie und Virologie; Verstehen; infectious diseases; Medicine/Public Health, general; Virology; Infectious Diseases; Immunology; Medical Microbiology; Health Sciences; Virology; Diseases; Immunology; Medizin, allgemein; Mikrobiologie (nicht-medizinisch); Krankheiten und Störungen; Immunologie; BC; EA
In multicellular organisms the establishment, maintenance, and programmed alterations of cell-type specific gene expression patterns are regulated by epigenetic mechanisms. Thus, epigenetic alterations (DNA methylation, DNA associated Polycomb-Trithorax protein complexes, histone modifications) ensure the unique transcriptional activity and phenotypic diversity of diploid cells that carry identical or nearly identical DNA sequences.
Because DNA methyltransferase I (DNMT1) associates with replication foci during S phase and prefers hemimethylated DNA as a substrate, DNMT1 ensures the clonal propagation of cytosine methylation patterns (maintenance methylation). Thus, DNA methylation may provide a memory function by helping progeny cells to “remember” their proper cellular identity.
modification of histone tails.
epigenetic alterations malignant tumors epigenetic dysregulation a wide spectrum of human diseases epigenetic memory via Epigenetic regulatory mechanisms.- Role of DNMT3B Mutations in the Pathogenesis of ICF Syndrome.- Dysfunction of the Methyl-Cpg Binding Protein Mecp2 in Rett Syndrome.- Epigenetic Alterations in Glioblastoma Multiforme.- Aberrant Epigenetic Regulation in Breast Cancer.- The Impact of Epigenetic Alterations on Diagnosis, Prediction and Therapy of Prostate Cancer.- Epigenetic Reprogramming in Lung Carcinomas.- Epigenetic changes in virus associated neoplasms.- Genetic and Epigenetic Determinants of Aggression.- Co-Regulation and Epigenetic Dysregulation in Schizophrenia and Bipolar Disorder.- Disruption of Epigenetic Mechanisms in Autoimmune Syndromes.- Imprinting Disorders.- Epigenetics of cardiovascular diseases.- Microbe-induced epigenetic alterations.In multicellular organisms the establishment, maintenance, and programmed alterations of cell-type specific gene expression patterns are regulated by epigenetic mechanisms. Thus, epigenetic alterations (DNA methylation, DNA associated Polycomb-Trithorax protein complexes, histone modifications) ensure the unique transcriptional activity and phenotypic diversity of diploid cells that carry identical or nearly identical DNA sequences.
Because DNA methyltransferase I (DNMT1) associates with replication foci during S phase and prefers hemimethylated DNA as a substrate, DNMT1 ensures the clonal propagation of cytosine methylation patterns (maintenance methylation). Thus, DNA methylation may provide a memory function by helping progeny cells to “remember” their proper cellular identity.
modification of histone tails.
epigenetic alterations malignant tumors epigenetic dysregulation a wide spectrum of human diseases epigenetic memory viaPatho-Epigenetics is a new discipline, and it’s covered here in detail
Written by experts in this exciting new field
Brings together recent results of epigenetic research with special attention to disease development
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